Fig. 5 | Scientific Reports

Fig. 5

From: AKR1C3 mediates gastric cancer cell invasion and metastasis via the AKT and JNK/p-NF-κB signaling pathways

Fig. 5

Analysis of the AKT, JNK, NF-κB, STAT3 signaling pathway mediating AKR1C3’s inhibition of EMT. Western blot analysis showing the expression of upstream proteins involved in EMT in both AGS and SNU216 cells with AKR1C3 overexpression or knockdown. *P < 0.05, unpaired t test, pCDH versus pAKR1C3. *P < 0.05, one-way ANOVA with Dunnett’s post hoc test, pSuper versus psh1AKR1C3 and pSuper versus psh2AKR1C3.

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