Extended Data Fig. 8: Systemic sustained hypoxia does not induce Aß deposition in wild-type mice.
From: Hypoxia compromises the mitochondrial metabolism of Alzheimer’s disease microglia via HIF1
a–c, 14-month-old wild-type mice were exposed to normoxia (N, 21% O2) or sustained hypoxia (H, 9% O2) for 21 days. No Thio-S (a), Aß (b) or pTAU (c) reactive deposits were found (n = 3 –N– or 4 –H– biological independent mice). Scale bars are 1 mm.
