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Author Correction: Smac mimetics and oncolytic viruses synergize in driving anticancer T-cell responses through complementary mechanisms

Dae-Sun Kim
Himika Dastidar
Douglas J. Mahoney
Amendments and CorrectionsOpen Access24 May 2018
Nature Communications

Volume: 9, P: 1
VSV^∆M51 drives CD8⁺ T cell-mediated tumour regression through infection of both cancer and non-cancer cells

The role of non-cancer cells infected by oncolytic viruses (OV) in cancer regression remains elusive. Here the authors engineer OV-sensitive and OV-resistant cancer cell lines and show that OV infection of non-cancer cells can elicit effective antitumour immunity via enhancing DC function and CD8⁺ T cell activation.

Jahanara Rajwani
Daniil Vishnevskiy
Douglas J. Mahoney
ResearchOpen Access15 Nov 2024
Nature Communications

Volume: 15, P: 1-20
Smac mimetics and oncolytic viruses synergize in driving anticancer T-cell responses through complementary mechanisms

Oncolytic viruses (OV) and second mitochondrial activator of caspase (Smac)-mimetic compounds (SMC) synergistically kill cancer cells directly. Here, the authors show that SMC and OV therapies combination also synergize in vivo by promoting anticancer immunity through an increase in CD8⁺ T-cell response.

Dae-Sun Kim
Himika Dastidar
Douglas J. Mahoney
ResearchOpen Access24 Aug 2017
Nature Communications

Volume: 8, P: 1-13

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