Nature Search

PTEN/MMAC1 is infrequently mutated in pT2 and pT3 carcinomas of the prostate

Jin-Tang Dong
Tavis W Sipe
Henry F Frierson Jr
Research14 Oct 1998
Oncogene

Volume: 17, P: 1979-1982
ZFHX3 is integral to androgen/AR signaling involving protein association with AR in prostate cancer cells

Xing Fu
Zhiqian Zhang
Jin-Tang Dong
ResearchOpen Access01 Jul 2025
Scientific Reports

Volume: 15, P: 1-11
Klf5 acetylation regulates luminal differentiation of basal progenitors in prostate development and regeneration

The role of the Klf5 in early and postnatal prostate development and in regeneration is unclear. Here, the authors show that Klf5 acetylation regulates and maintains luminal differentiation of prostate basal progenitors and is essential following androgen-induced regeneration.

Baotong Zhang
Xinpei Ci
Jin-Tang Dong
ResearchOpen Access21 Feb 2020
Nature Communications

Volume: 11, P: 1-16
Frequent somatic mutations of the transcription factor ATBF1 in human prostate cancer

Xiaodong Sun
Henry F Frierson
Jin-Tang Dong
Research06 Mar 2005
Nature Genetics

Volume: 37, P: 407-412
LEM4 confers tamoxifen resistance to breast cancer cells by activating cyclin D-CDK4/6-Rb and ERα pathway

Tamoxifen resistance is a major problem in the treatment of ERa positive breast cancer. Here, the authors show that LEM4 confers tamoxifen resistance by activating the cyclin D-CDK4/6-Rb axis and ERa signaling in endocrine-resistant breast cancer.

Ang Gao
Tonghua Sun
Zhengmao Zhu
ResearchOpen Access09 Oct 2018
Nature Communications

Volume: 9, P: 1-17
A possible tumor suppressor role of the KLF5 transcription factor in human breast cancer

Ceshi Chen
Hina V Bhalala
Jin-Tang Dong
Research23 Sept 2002
Oncogene

Volume: 21, P: 6567-6572
Acetylation of KLF5 maintains EMT and tumorigenicity to cause chemoresistant bone metastasis in prostate cancer

The therapies for bone metastatic prostate cancer are limited and the underlying mechanisms are unclear. Here, the authors show that bone derived TGF-β induces acetylation of KLF5 (Ac-KLF5), and Ac-KLF5 promotes prostate cancer bone metastasis and resistance to docetaxel by upregulating CXCR4.

Baotong Zhang
Yixiang Li
Jin-Tang Dong
ResearchOpen Access17 Mar 2021
Nature Communications

Volume: 12, P: 1-21
An 18-gene signature of recurrence-associated endothelial cells predicts tumor progression and castration resistance in prostate cancer

Bing-Biao Lin
Qingqing Huang
Jun Pang
ResearchOpen Access12 Jul 2024
British Journal of Cancer

Volume: 131, P: 870-882
TTK promotes mesenchymal signaling via multiple mechanisms in triple negative breast cancer

Jamie L. King
Baotong Zhang
Jin-Tang Dong
ResearchOpen Access12 Sept 2018
Oncogenesis

Volume: 7, P: 1-13
ZFHX3 is indispensable for ERβ to inhibit cell proliferation via MYC downregulation in prostate cancer cells

Qingxia Hu
Baotong Zhang
Jin-Tang Dong
ResearchOpen Access12 Apr 2019
Oncogenesis

Volume: 8, P: 1-15
Ubiquitin–proteasome degradation of KLF5 transcription factor in cancer and untransformed epithelial cells

Ceshi Chen
Xiaodong Sun
Jin-Tang Dong
Research21 Feb 2005
Oncogene

Volume: 24, P: 3319-3327

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PTEN/MMAC1 is infrequently mutated in pT2 and pT3 carcinomas of the prostate

ZFHX3 is integral to androgen/AR signaling involving protein association with AR in prostate cancer cells

Klf5 acetylation regulates luminal differentiation of basal progenitors in prostate development and regeneration

Frequent somatic mutations of the transcription factor ATBF1 in human prostate cancer

LEM4 confers tamoxifen resistance to breast cancer cells by activating cyclin D-CDK4/6-Rb and ERα pathway

A possible tumor suppressor role of the KLF5 transcription factor in human breast cancer

Acetylation of KLF5 maintains EMT and tumorigenicity to cause chemoresistant bone metastasis in prostate cancer

An 18-gene signature of recurrence-associated endothelial cells predicts tumor progression and castration resistance in prostate cancer

TTK promotes mesenchymal signaling via multiple mechanisms in triple negative breast cancer

ZFHX3 is indispensable for ERβ to inhibit cell proliferation via MYC downregulation in prostate cancer cells

Ubiquitin–proteasome degradation of KLF5 transcription factor in cancer and untransformed epithelial cells

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