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Cell Death and Differentiation (2)
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Showing 1–7 of 7 results

Advanced filters: Author: Robert Nechanitzky Clear advanced filters

Author Correction: Fcmr regulates mononuclear phagocyte control of anti-tumor immunity

Shawn P. Kubli
Larsen Vornholz
Tak W. Mak
Amendments and CorrectionsOpen Access03 Sept 2019
Nature Communications

Volume: 10, P: 1
Cholinergic regulation of thymocyte negative selection

Acetylcholine–α9 nAChR signaling regulates thymic negative selection.

Shaofeng Liu
Chunxing Zheng
Tak W. Mak
Research21 May 2025
Nature Immunology

Volume: 26, P: 881-893
Fcmr regulates mononuclear phagocyte control of anti-tumor immunity

Myeloid cells modulate the immune response within the tumour microenvironment, but the underlying mechanisms remain largely unknown. Here, the authors show that Fcmr – the putative receptor for soluble IgM – is a potent negative regulator of anti-tumour immunity.

Shawn P. Kubli
Larsen Vornholz
Tak W. Mak
ResearchOpen Access18 Jun 2019
Nature Communications

Volume: 10, P: 1-15
CaSSiDI: novel single-cell “Cluster Similarity Scoring and Distinction Index” reveals critical functions for PirB and context-dependent Cebpb repression

Robert Nechanitzky
Parameswaran Ramachandran
Tak W. Mak
ResearchOpen Access21 Feb 2024
Cell Death & Differentiation

Volume: 31, P: 265-279
Cholinergic control of Th17 cell pathogenicity in experimental autoimmune encephalomyelitis

Robert Nechanitzky
Duygu Nechanitzky
Tak W. Mak
Research17 Dec 2022
Cell Death & Differentiation

Volume: 30, P: 407-416
Tumor-specific cholinergic CD4⁺ T lymphocytes guide immunosurveillance of hepatocellular carcinoma

Zheng et al. show that ChAT-expressing cholinergic T cells suppress the development of liver cancer via their cholinergic activity, which constrains calcium/NFAT signaling induced by TCR engagement.

Chunxing Zheng
Bryan E. Snow
Tak W. Mak
ResearchOpen Access28 Aug 2023
Nature Cancer

Volume: 4, P: 1437-1454
Transcription factor EBF1 is essential for the maintenance of B cell identity and prevention of alternative fates in committed cells

Lineage-commitment factors enforce irreversible loss of alternative lineage potentials during development. Grosschedl and colleagues show that EBF1 expression in committed pro-B cells suppresses T cell and innate lymphoid cell potential.

Robert Nechanitzky
Duygu Akbas
Rudolf Grosschedl
Research30 Jun 2013
Nature Immunology

Volume: 14, P: 867-875