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Scientific Reports (3)
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Showing 1–6 of 6 results

Advanced filters: Author: Xiue Ma Clear advanced filters

CDC-like kinase 4 deficiency contributes to pathological cardiac hypertrophy by modulating NEXN phosphorylation

Phosphorylation catalyzed by kinases is a key event in signaling pathways involved in cardiomyocyte hypertrophy. Here the authors show that the kinase CLK4 ameliorates cardiac hypertrophy by phosphorylating NEXN.

Jian Huang
Luxin Wang
Yi-Han Chen
ResearchOpen Access30 Jul 2022
Nature Communications

Volume: 13, P: 1-13
Discovery of broad-spectrum fungicides that block septin-dependent infection processes of pathogenic fungi

Very-long-chain fatty acids (VLCFAs) mediate recruitment of septins to the plasma membrane in fungal appressoria, leading to formation of a septin ring at the point of plant infection. VLCFA biosynthesis-inhibiting chemicals prevent septin assembly and have broad-spectrum fungicidal activity.

Min He
Jia Su
Xuewei Chen
Research21 Sept 2020
Nature Microbiology

Volume: 5, P: 1565-1575
LRP6 acts as a scaffold protein in cardiac gap junction assembly

LRP6 is known for its role as a Wnt co-receptor essential for the canonical Wnt/β-catenin signaling. Here, Li et al. show that LRP6 exerts a Wnt-independent scaffold function and regulates connexin 43 gap junction formation and coupling of cardiomyocytes in mouse hearts.

Jun Li
Changming Li
Yi-Han Chen
ResearchOpen Access02 Jun 2016
Nature Communications

Volume: 7, P: 1-11
Generation of gene-modified goats targeting MSTN and FGF5 via zygote injection of CRISPR/Cas9 system

Xiaolong Wang
Honghao Yu
Yulin Chen
ResearchOpen Access10 Sept 2015
Scientific Reports

Volume: 5, P: 1-9
Validation of SinoSCORE for isolated CABG operation in East China

Xiue Ma
Yunqian Wang
Yangyang Zhang
ResearchOpen Access01 Dec 2017
Scientific Reports

Volume: 7, P: 1-9
Cecropin B Represses CYP3A29 Expression through Activation of the TLR2/4-NF-κB/PXR Signaling Pathway

Xiaoqiao Zhou
Xiaowen Li
Dingren Bi
ResearchOpen Access14 Jun 2016
Scientific Reports

Volume: 6, P: 1-14